Sochalska Maja, |
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University / Clinic: Medical University Innsbruck |
Institute: Department of Developmental Immunology | |
Research Area: cell death signaling, role and function of anti-apoptotic Bcl-2 family members in normal development | |
Email: maja.sochalska@i-med.ac.at | |
Web: | |
Funded by: FWF | |
Research Topic: | |
dormeo дормео |
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Selected publications: | |
Sochalska M, Ottina E, Tuzlak S, Herzog S, Herold M, Villunger A Conditional knockdown of BCL2A1 reveals rate-limiting roles in BCR-dependent B-cell survival. Cell Death Differ. 2016 Apr;23(4):628-39. doi: 10.1038/cdd.2015.130. PubMed PMID: 26450454; PubMed C |
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Sochalska M, Schuler F, Weiss J, Prchal-Murphy M, Sexl V, Villunger A MYC selects against reduced BCL2A1/A1 protein expression during B cell lymphomagenesis Oncogene. 2016 Oct 3. doi: 10.1038/onc.2016.362. [Epub ahead of print] PubMed PMID: 27694901 |
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Ottina E, Sochalska M, Sgonc R, Villunger A The BH3-only protein Bad is dispensable for TNF-mediated cell death. Cell Death Dis. 2015 Jan 22;6:e1611. |
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Sochalska M, Tuzlak S, Egle A, Villunger A Lessons from gain- and loss-of-function models of pro-survival Bcl2 family proteins: implications for targeted therapy. FEBS J. 2015 Mar;282(5):834-49. |
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Ottina E, Lyberg K, Sochalska M, Villunger A, Nilsson G Knockdown of the antiapoptotic Bcl-2 family member A1/Bfl-1 protects mice from anaphylaxis. J Immunol. 2015 Feb 1;194(3):1316-22. |
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