Sochalska Maja,

University / Clinic: Medical University Innsbruck
Institute: Department of Developmental Immunology
Research Area: cell death signaling, role and function of anti-apoptotic Bcl-2 family members in normal development


  Funded by: FWF

Research Topic:

Selected publications:
  Sochalska M, Ottina E, Tuzlak S, Herzog S, Herold M, Villunger A
Conditional knockdown of BCL2A1 reveals rate-limiting roles in BCR-dependent B-cell survival.
Cell Death Differ. 2016 Apr;23(4):628-39. doi: 10.1038/cdd.2015.130. PubMed PMID: 26450454; PubMed C

  Sochalska M, Schuler F, Weiss J, Prchal-Murphy M, Sexl V, Villunger A
MYC selects against reduced BCL2A1/A1 protein expression during B cell lymphomagenesis
Oncogene. 2016 Oct 3. doi: 10.1038/onc.2016.362. [Epub ahead of print] PubMed PMID: 27694901

  Ottina E, Sochalska M, Sgonc R, Villunger A
The BH3-only protein Bad is dispensable for TNF-mediated cell death.
Cell Death Dis. 2015 Jan 22;6:e1611.

  Sochalska M, Tuzlak S, Egle A, Villunger A
Lessons from gain- and loss-of-function models of pro-survival Bcl2 family proteins: implications for targeted therapy.
FEBS J. 2015 Mar;282(5):834-49.

  Ottina E, Lyberg K, Sochalska M, Villunger A, Nilsson G
Knockdown of the antiapoptotic Bcl-2 family member A1/Bfl-1 protects mice from anaphylaxis.
J Immunol. 2015 Feb 1;194(3):1316-22.